Though the Google Machine has pissed me off in the past, I still loves it. One thing I really like about teh Google is Google News, where I can pop in words Im interested in, and read the latest news on that keyword every day.
No big surprise one of my keywords is ‘endogenous retrovirus’.
Well today, under the ‘ERV’ tab, I noticed an article by Adnanabannabiingbong Yahya, or someone writing under his name:
QUESTION: How do you account for Endogenous Retro Viruses (ERVs), and the markers they have left in inheritable genes? You can clearly “trace” ancestry using these ERV markers. How do you explain these markers that are the same in Human and Chimp DNA? The odds of this happening as many times as they have are mind-boggling. How do you explain how well it can be traced down a phylogenetic tree? How can this be explained by anything but common ancestry?
ADNAN OKTAR: Darwinists have the tendency to interpret ERVs (endogenous retroviruses) as a heretic legacy from our imaginary ape forebears. They claim that retroviruses, which make up 8 percent of human genome, were active in the past but, undergoing some mutations, lost their functions, and were left to human genome from the imaginary ape forebear as junk DNA.
First; ERVs exist not only in chimpanzee genome but in the genome of all mammals living all over the world. Presenting ERVs’ presence in chimpanzee as an evidence for alleged human evolution is a serious defeat of logic.
Second; according to Darwinists ERVs should be junk inherited from imaginary ape forebears. However, new findings have refuted this important Darwinist claim. ERVs have functions and assume three important roles in human cell. 1) gene regulation 2) formation of placenta 3) formation of disorders such as cancer, infertility. That ERVs are not junk DNA definitely eliminates all Darwinist claims.
Yes, thats nice. All of that is covered under ‘Index to Creationist Claims about ERVs‘. Nothing new at all.
EXCEPT this part: “However, new findings have refuted this important Darwinist claim. ERVs have functions and assume three important roles in human cell. 1) gene regulation 2) formation of placenta 3) formation of disorders such as cancer, infertility.”
Long-time readers of ERV will be familiar with that ‘infertility’ comment. I think I probably brought it up first when PZ and I were discussing ‘epigenetics’ and ‘ERVs’ on BloggingHeads last year (~minute 11). Youll also note that, at the time I said it, I also said that epigenetic disregulation and ERVs causing spontaneous abortions, perhaps even forms of infertility, were just a pet theory of mine that I had no evidence for. I brought it up again here.
Look up “ERV + infertility” in PubMed. Theres nothin. “Retrovirus + infertility” is all how to prevent HIV-1 transmission, and gene therapy experiments. “”Endogenous retrovirus” + infertility” will get you one hit from Japan that talks about homologous recombination of a HERV might be associated with gimpy sperm. Thats it.
And yet, curiouser and curiouser, Haruynamalakahiki responded to a ‘question’ using that personal pet theory of mine.
Edit: *sad panda* Harunny Poopies wasnt reading my blag, he was plagiarizing Answers in Genesis:
Contrary to being “junk” DNA, HERVs are thought to play at least three major roles. One role is to control the regulation of genes (the expression of proteins from genes). Members of the HERV-K family are typically found in areas near genes. The regulatory role of HERVs has been demonstrated in the liver, placenta, colon, and other locations. It was recently reported that an endogenous retrovirus in sheep was necessary for maintaining pregnancy, as it was important in the formation of the placenta. HERVs also play a role in disease, and have been linked to various cancers and male infertility.
‘Male infertility’ is just homologous recombination of an ERV cutting out a gene. Not anything nearly as cool as ERVs coming back to life to ravage an embryo.